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Interest is real, but the responsible approach is not to treat it as a shortcut

The persistent limitation is translational: rodent lipid handling differs from human physiology, and even the best diet models only partially recapitulate the slow, heterogeneous fibrotic progression seen in human MASH which is why an animal liver-fat signal is treated as hypothesis-generating rather than confirmatory

One last article was eliminated in final analysis because the laboratory methodology and pathogenesis of periodontal disease are now refuted by more recent scientific evidence

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