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ATP depletion stimulates PKC-alpha activity and leads to translocation of the enzyme to the plasma membrane, thereby enhancing serine phosphorylation of membrane proteins, such as the Ca 2+ -permeable cation channel (Fig
Mol Carcinog (2019) 58:220717

lumbricoides -infected pregnant women

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Thiol groups are strong reducing agents thus glutathione serves as an electron donor and in the process is converted to its oxidised form

The probable pathway likely involves PCs augmenting GLUT4 translocation and glucose uptake in skeletal muscle through an AMPK-dependent mechanism, subsequently enhancing GLP-1activated insulin signaling and lowering postprandial hyperglycemia
