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Growth hormone versus placebo treatment for one year in growth hormone deficient adults: increase in exercise capacity and normalization of body composition

Enhanced -synuclein toxicity is epistatically regulated by the PARK6 homolog, pink-1 Given that altered proteasome, protein mishandling, and GSH deficiencies associate with mitochondrial dysfunction in sporadic PD, 11, 20, 21, 32, 36 we sought to understand how metabolite-induced proteasome inhibition relates to PINK1 and Parkin

doi: 10.1016/j.cmet.2022.08.017

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Many of these studies have not shown significant decreases in homocysteine with the T allele, important because low homocysteine would be evidence of increased CBS activity