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Reciprocal regulation between SIRT6 and mir-122 controls liver metabolism and predicts hepatocarcinoma prognosis

Chronic metabolic stress induces beta cells to adopt a senescence-associated secretory phenotype (SASP), characterised by cytokines and ECM remodelling factors, that spreads into the surrounding environment promoting beta cell failure [57]

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However, in the presence of oxidative or nitrosative stress, GST can form homodimers that alter the reduced states of cysteine residues in its structure, resulting in JNK dissociation from the GSTJNK heterocomplex and causing subsequent activation of the c-Jun protein

oleifera extracts on cell lines: Methanolic leaf extract applied to HBF4 cells for 24 h caused noticeable changes at doses 1000 g/mL, including increased cell size