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While antioxidants such as vitamin E were once considered protective against cancer, the Selenium and Vitamin E Cancer Prevention Trial (SELECT) paradoxically revealed an increased risk of prostate cancer, 4 leading to an emerging view that ferroptosis inhibition by dietary antioxidants may underlie such outcomes

Translational Andrology and Urology

2012), but this was neither mimicked by the supplementation of control cells with antioxidant products of HO-1 nor by antioxidant N-acetylcysteine

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The patients receiving drug treatment reported a decrease in knee pain and swelling of the knee joint as well as increased knee flexion and walking distance (Kimmatkar et al., 2003)

Methionine restriction induces apoptosis of prostate cancer cells via the c-Jun N-terminal kinase-mediated signaling pathway