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GH exerts autocrine/paracrine effects by binding to GHR in the same or a neighboring cell surface and initiating JAK2- and SRC-mediated signaling cascades, which decrease pro-apoptotic molecules (Bax and PPAR) and suppress caspase protein activation (Figure 4A)

Faderl S, Ferrajoli A, Ravandi F, Verstovsek S, Wierda W, Borthakur G , Thomas DA, Cortes J, Kornblau S, Estrov Z, Andreeff M, Beran M, Giles F, Huang X, Kwari M, Kantarjian H

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\n\n\n\n The Gap Between Animal Research and Human Clinical Trials \n\n\n\n One of the most important caveats in evaluating the research on BPC-157 for tendon healing is the substantial gap between the robustness of preclinical evidence and the current absence of large-scale, peer-reviewed human clinical trials

A cosmetic risk would be (for lack of a better descriptor) uneven lumpiness as the fat/fat stem cells may not be evenly distributed or could grow in a not entirely even way