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DAQs could conjugate with and modify Cys95 and Cys253 residues of Parkin protein, leading to its insolubility, as revealed in post-mortem patient brain samples and in PD models in vivo [82, 134]

We also have murine models of calcification that we use to obtain fate maps about potential progenitor populations contributing to cardiac calcification and study mechanisms driving progenitors to adopt an osteoblast (calcium forming cell) fate

Therefore, we advocate incorporating Eadie-Hofstee transformation into routine kinetic analyses, especially for nanozymes composed of heterogeneous structures or composite materials, where active-site diversity likely influences catalytic performance

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