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Mechanistically, inhibition of CD38 in vivo via apigenin leads to an increase in cellular NAD levels and activation of NAD-dependent enzymes, (e.g.SIRTUINs) [72]

The search plan was made in Medline for Pubmed with the following mesh terms: IGF-1 and metabolism, carbohydrate, lipids, proteins, amino acids, metabolic syndrome, cardiovascular disease, diabetes between the years 19632015

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Administration can occur simultaneously or separately based on convenience