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Additionally, the mechanism by which PNPLA3 inhibited lipolysis involved the re-esterification of mobilized FAs (Figure 1)

This is because hypokalemia and thrombocytosis could occur due to the increase in erythrocyte metabolism following vitamin B12 therapy

Accumulation of PNPLA3 on lipid droplets is the basis of associated hepatic steatosis

These effects create a powerful metabolic shift toward fat utilization and energy expenditure

While this provides body composition benefits including fat loss and muscle growth, it also affects glucose metabolism (often elevating blood sugar), increases IGF-1 (with implications for cell proliferation), and costs significantly more than AOD-9604

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