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In this review, we focus on the ETC as a source of ROS and its modulation by oncogenic pathways, which generates a vicious cycle that resets ROS levels to a higher homoeostatic set point, sustaining the cancer cell phenotype

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Normothermic Ex Vivo Kidney Perfusion for the Preservation of Kidney Grafts Prior to Transplantation

10.1007/s11883-012-0291-7 [DOI] [PMC free article] [PubMed] [Google Scholar] 40

However, despite the absence of ferritinophagy we found that intracellular ferrous iron levels in IB3-1 cells (but not C38 cells) increased following erastin and FAC exposure, which requires further investigation as the pathways responsible for this increase in redox active iron in response to ferroptotic induction are currently unknown, but will become targets for future treatment interventions