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van Eijkeren, R

W.et al (2012)

Virus excites intracellular ROS accumulation, which triggers cell death by cellular ROS sensors (KEAP1), PGAM5 phosphorylation, and the pro-apoptotic factor AIFM1

These two properties together are exceedingly rare in neuroprotective compounds and open the door to practical administration protocols that do not require injections or intranasal delivery

Seizure activity results in calcium- and mitochondria-independent ROS production via NADPH and xanthine oxidase activation

Neuroimaging studies employing magnetic resonance imaging have explored the relationship between white matter structure and cognitive impairment in patients who underwent a chemotherapy regimen comprising uracil, cyclophosphamide, methotrexate, and 5-fluorouracil after surgery, comparing the results with those of patients who did not receive chemotherapy