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Upregulation of LOX and LOXL2 has been implicated in several pathologies, including cancer, cardiac fibrosis, idiopathic pulmonary fibrosis, vascular stiffening in aging, and pulmonary hypertension 8,9,10,11,12,13,14

and NIASKI, W

(2013) applied FTIR to analyze B

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Distinct Nrf1/2-independent mechanisms mediate As 3+-induced glutamate-cysteine ligase subunit gene expression in murine hepatocytes