hypothesis glutathione persister cells FSP1 and histone deacetylases suppress cancer cell ferroptosis Sublethal cytochrome c release generates

untreated) and AUC (~ 38% vs

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doi:10.1016/S0026-0495(98)90055-5

These properties allow peroxynitrite to significantly alter the function of a considerable number of proteins, to degrade membrane structure by peroxidizing lipids, to turn off crucial metabolic functions within mitochondria, and to inflict serious damage to nucleic acids, activating a major pathway of cell injury and inflammation orchestrated by the nuclear enzyme PARP

doi: 10.1038/srep35645 (2016)