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PubMed Central , For your reference: For your reference: For your reference: 15, 25 Mateus, Filipa G., et al

Cellular & Molecular Biology Letters

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Although the precise pathogenic mechanisms of HCV-3 mediated steatosis are still unknown, HCV-3 modulates host lipid metabolism and appears to influence unique mechanisms of fat metabolism and transportation within the liver including microsomal triglyceride transfer protein (MTTP), sterol regulatory element binding protein 1c (SREBP-1c), and peroxisome proliferator associated receptor alpha (PPAR-)[20]

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CD36-mediated ferroptosis dampens intratumoral CD8(+) T cell effector function and impairs their antitumor ability
