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Excessive metabolization of ATP to ADP may determine the course of inflammation

[DOI] [PMC free article] [PubMed] [Google Scholar] 102.Mazzulli J.R., Xu Y.H., Sun Y., Knight A.L., McLean P.J., Caldwell G.A., Sidransky E., Grabowski G.A., Krainc D

However, a recent report demonstrated a non-apoptotic function of Bax in long-term depression of synaptic transmission with caspase-3 activation and Bax modulation as pivotal elements during synaptic plasticity [45]