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In the stasis model, neutrophils lacking TLR9 release more NETs, which contribute to venous thrombosis formation (173)

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found that significant enrichment of Th2 and Th17 lymphocyte subsets identified within bronchoalveolar lavage samples obtained from individuals diagnosed with asthma could trigger IL-4 overproduction and subsequently augment the capability of inducing glucocorticoid resistance in vitro [131]

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Various modulators of ferroptosis, including inhibitors and inducers, provide novel perspectives for addressing bone and joint disorders
