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[DOI] [PubMed] [Google Scholar] 10.Li A, Wu S, Li Q, Wang Q, Chen Y

Blocking Silicosis Fibrosis by Targeting Fibroblasts Pulmonary fibrosis is characterized by excessive secretion of collagen, fibronectin, and elastin by myofibroblasts, leading to an imbalance in MMP/TIMP regulation and accumulation of the ECM [84]

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Molecules such as fibroblast growth factor 21 (47), BCL2 interacting protein 3/NIP3-like protein X (48) and acteoside (via the NRF2-mitophagy axis) (49) mitigate ferroptosis by restoring mitochondrial function or removing damaged mitochondria, underscoring the protective role of mitophagy against ferroptosis

Ginsenoside Rh1 exerts neuroprotective effects by activating the PI3K/Akt pathway in amyloid- induced SH-SY5Y cells

Heitzer T, Just H, Mnzel T