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The underlying mechanism involves a shift in the balance of dopamine D1- and D2-receptor expression in the striatum

The results should not be construed as financial advice

The study reports that D1/D5 dopamine receptor signaling and small conductance calcium-activated potassium channels contribute to high concentration catecholamine induced endothelial cell dysfunction

Proc Am Assoc Cancer Res 45((#872)):20, 2004
