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Description
Identification and characterization of the primary demyelinating lesion

That increases an adaptive capability via preadaptation/hormetic mechanisms

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Deficits in E2-dependent control of feeding, weight gain, and cholecystokinin satiation in ER- null mice
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The detection of a molecular weight peak at m/z = 573 in PM (Lid/AG = 1/1) suggests that Lid and AG interacted in the solution

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