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Description
doi: 10.1158/1078-0432.CCR-05-1830
Intranasal delivery of copper oxide nanoparticles induces pulmonary toxicity and fibrosis in C57BL/6 mice
A.StrathdeeG.SluddenJ.KayeS
This process is driven by disrupted GSH homeostasis, resulting from diminished GSH synthesis and impaired activity of two critical antioxidant systems: (1) the cystine/glutamate antiporter xCT, which imports cystine to fuel GSH production and (2) GPX4, which utilizes GSH to convert lipid peroxides into less toxic lipid alcohols (Stockwell et al., 2017
labeling and storage requirements depend on the manufacturer
opioid) or level of severity
