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doi:10.1210/jc.2018-00448
bioRxiv [Internet]
other stuff' section of the OAuth integration setup page in AdvocateHub
These metabolic disorders include changes in glucose and FA transport from the extracellular environment into the cytoplasm, anaerobic glycolysis in the cytoplasm, and mitochondrial oxidative phosphorylation (Zhi et al., 2016
Chem Biol Interact (2006) 160:140
Offspring, which is compound heterozygous for the leptin receptor defect, develops the obese phenotype (O-ZSF1), and eventually HFpEF
