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Shown are IC 50 normalized to cisplatin alone (dotted line at y = 1) (n = 6 technical replicates from two independent experiments)

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In the TMLHE gene knockout mouse model, the carnitine biosynthetic pathway is inhibited due to the loss of TMLD activity in the liver, affecting the availability of carnitine in the heart, significantly reducing the rate of synthesis of LCACs, and contributing to the protection of mitochondrial function after myocardial IRI [215]

Carnitine also interacts with the anticonvulsants valproic acid, phenobarbital, phenytoin, or carbamazepine

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