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A prominent alternative viewpoint argues that the increased glucose uptake observed during neural activation is primarily metabolized via aerobic glycolysis (non-oxidative glucose consumption) (Fox et al., 1988) within the activated tissue itself, producing lactate that is largely released rather than shuttled to neurons

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TST degrades ROS with the help of TXN in cell-free systems and suppresses oxidative stress in the liver of animals exposed to radiation [[58], [59], [60], [61]]