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4.1 Pharmacokinetic and pharmacodynamic considerations of antioxidant supplementation in AMS As mentioned above, oxidative stress has been proposed as a key contributor to the pathophysiology of AMS, driven by hypobaric hypoxiainduced mitochondrial dysfunction, increased ROS generation, endothelial activation, and neurovascular dysregulation

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The final solution was lyophilized to obtain GSH-CDs-Ce6

Intestinal barrier function and immune homeostasis are missing links in obesity and type 2 diabetes development