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PlexinB1/B2 activation promotes nuclear trafficking of both AR [33] and GR [34] as well as the NF-kB subunit, p65 [35] (Supplementary Fig

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This interaction inhibits Keap1-mediated ubiquitination and degradation of NRF2, resulting in the upregulation of antioxidant genes and contributing to chemotherapy resistance in NSCLC [100]

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