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The results indicated a reduction in levels of ROS, MDA, and iron in the blood, an increase in the expression of GSH and GPX4, and alleviation of oxidative stress-induced mitochondrial damage in the spinal cord
Intestinal permeability and systemic endotoxemia in patients with acute pancreatitis

The STAT1/HMGB1/NF-B pathway in chronic inflammation and kidney injury after cisplatin exposure
Sugano K, Tack J, Kuipers EJ, Graham DY, El-Omar EM, Miura S, Haruma K, Asaka M, Uemura N, Malfertheiner P (2015) Kyoto global consensus report on Helicobacter pylori gastritis

Lopez-Erauskin and colleagues postulated a model, in which an excess of C26:0 induces mitochondrial production of reactive oxygen species leading to mitochondrial oxidative damage to DNA and proteins, inefficient oxidative phosphorylation and finally to dying-back axonopathy [52]

He L, He T, Farrar S, Ji L, Liu T, Ma X (2017) Antioxidants maintain cellular redox homeostasis by elimination of reactive oxygen species
