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The sequencing reads were deduplicated using umi tools (v1.1.6), mapped to the reference plasmid using BWA-MEM, and further cleaned using fgbio (v2.4.0) to remove the soft-clip sequences

A breakdown in metabolic reprogramming causes microglia dysfunction in Alzheimer's disease

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Tanycytic VEGF-A boosts bloodhypothalamus barrier plasticity and access of metabolic signals to the arcuate nucleus in response to fasting

AGEs, which form through non-enzymatic reactions between glucose and proteins, engage receptors like RAGE, activating nuclear factor kappa B ( NF-B ) pathways and further promoting ROS production and inflammatory cytokine release [22]

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