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Furthermore, in vitro studies on C2C12 cells confirm that AX promotes myoblast differentiation and the formation of new mitochondria, underscoring the key role of mitochondrial biogenesis as a mechanism through which AX improves skeletal muscle function and recovery in obese conditions [12]

Retrieved on December 22, 2019, from Solis, C

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Importantly, TMA, TMAO, and FMO3 were all decreased with -sitosterol treatment, indicating that its effects may not be solely due to TMA lyase inhibition

Molecular mechanisms associated with diabetic endothelial-erectile dysfunction

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