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The decreased regeneration capacity of muscles is due to both extrinsic (low amino acid intake and lack of muscle stimulation due to low physical activity) and intrinsic factors (blunted protein synthesis responses, and increased circulation of inflammatory and catabolic cytokines) [36]

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(2001) [86] reported that L-Carnitine, acting as a cofactor in fatty acid transport, may have a reparative effect by eliminating elevated intracellular levels of toxic acetyl-coenzyme A or by replacing fatty acids in the phospholipid membrane

Targeting lipid metabolism has emerged as a promising therapeutic strategy to correct immune dysfunction in these conditions

Role of carnitine esters in brain neuropathology