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The protective role of selenium metabolism is thought to be exerted through the neutralization of ROS via antioxidant activities of selenoproteins including GPXs and TXNRDs

However, discrete analysis of GH pulsatile parameters revealed that mean GH pulse amplitude and total GH total peak area may largely underlie this effect

Ferritin accumulation in dystrophic microglia is an early event in the development of Huntingtons disease

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V., Kalia, S

[50] found that activated ERK1/2 played a pro-apoptotic role by inducing BAX expression, MMP depolarization, mitochondrial Cyto-C release and caspase3 upregulation, which was in line with our findings