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ChIP-seq analysis revealed that PHF3-GFP bound to expressed genes and that recruitment to chromatin was reduced upon deletion of the SPOC domain, indicating that the interaction with phosphorylated Pol II CTD is critical for PHF3 binding to chromatin (Supplementary Fig

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Tafuri A, Andreeff M

In addition, evaluating inhibitory tonus as a measure of repressed plasticity in the post-injured brain, concomitant changes in the excitatory input may occur, making it difficult to estimate changes in the overall excitatory/inhibitory balance of the neural circuits undergoing rearrangement