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Focal loss of the glutamate transporter EAAT2 in a transgenic rat model of SOD1 mutant-mediated amyotrophic lateral sclerosis (ALS)

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Mutation spectrum of ATP7A, the gene defective in Menkes disease

Additionally, macrophages can directly impede preadipocyte differentiation and stimulate the expression of pro-fibrotic factors such as -smooth muscle actin (-SMA), prompting a myofibroblast-like phenotype, an effect that is potentiated by excess glucocorticoid exposure (193)

Redox stress An imbalance between oxidants and antioxidants in favour of the oxidants, leading to a disruption of redox signalling and control and/or molecular damage

These conflicting data need to be further validated in different diseases