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The unresolved questions surrounding the effect of cuprizone treatment on brain copper levels and complex chemistry make it difficult to judge if the copper chelating property of cuprizone plays a role in cuprizone-induced demyelination

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In experimental models of vascular injury, BPC-157 has been associated with the restoration of microcirculation and the prevention of pathological thrombosis

One posited mechanism is the activation of phospholipase C (PLC), which, according to some researchers, might subsequently facilitate the release of inositol triphosphate (IP3) and diacylglycerol (DAG)