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Transcriptional evidence for the role of chronic venlafaxine treatment in neurotrophic signaling and neuroplasticity including also glutatmatergic- and insulin-mediated neuronal processes

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Their transient knockdown, except CoQ8 and CoQ6 , reduced CoQ 10 levels, but just CoQ7 knockdown could hamper mitochondrial respiration and elevated ubiquinol to ubiquinone ratios and also cause the accumulation of a putative biosynthetic intermediate characterized by reversible redox property, like CoQ 10 (Yen et al., 2016)

and Moorthy B., Role of Cytochrome P450s in the Generation and Metabolism of Reactive Oxygen Species, Current Opinion in Toxicology 7 (2018): 4451

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