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These are collectively referred to as parkinsonism (1), and they are the result of the progressive loss of dopaminergic neurons in substantia nigra (2) along with the formation of Lewy bodies that comprise misfolded -synuclein in the neurons (3)

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Bostock, I, Antonoff, MB

2-Hydroxy-4-Methylselenobutanoic Acid Treatment Upregulated the Expression of Selenoproteins and Improved the Antioxidative Capacity of Porcine Intestinal Epithelial Cells To assess whether HMSeBA treatment affects the expression of selenoproteins and the antioxidant capacity of IPEC-J2 cells, we detected the mRNA and protein levels of selenoprotein and activities of antioxidative markers