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Giridharan N., Katlowitz K

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nor were found susceptible to extraneous stressors, highlighting that Sp exposure did not compromise organismal response to electrophiles (Fig

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AGE-1 converts the phospholipid PIP 2 into the second messenger PIP 3, whose elevated levels activate the 3-phosphoinositide dependent protein kinase-1 (PDK1) [11] and the protein kinases B (PKB12), thus leading to the phosphorylation of DAF-16, a homolog of the mammalian FoxO family of transcription factors [7,8]

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