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Activation of the intrinsic coagulation cascade leads to inappropriate thrombolytic events as well as resulting in increases in inflammatory cytokine release from leukocytes that are activated as a result of the pro-coagulant state

Moreover, if two modes of cell death occur concurrently, it remains important to determine the proportion each contributes to overall cell death, and whether they are linked by shared regulatory factors or are sequentially activated in a temporal order

Studies show it can act as both a NO promoter and stabilizer depending on the physiological context, helping maintain optimal blood flow to injured areas