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The pathophysiological mechanisms underlying smoking-induced endothelial dysfunction are intricate

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Treatment of HAECs with nicotine fostered intracellular ROS production, and this was buffered by N-acetyl-cysteine (NAC), a ROS inhibitor (Fig

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Headache 56(3):511518 Sedlic M, Mahovic D, Kruzliak P (2016) Epidemiology of primary headaches among 1,876 adolescents: a cross-sectional survey

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