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An increase in acetylated histone H4 was found in current smokers, while patients with COPD who had stopped smoking had increased histone H3 acetylation, suggesting that the persistent inflammation in the lungs of the COPD patients after smoking cessation may be regulated by histone H3 acetylation [29]

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[10] Signs and symptoms [edit] On average, exposures at 100 ppm or greater is dangerous to human health

Toxicol Appl Pharmacol

In addition, abnormal proliferative hepatocytes and precancerous cells cannot be killed, because of hepatitis viruses-mediated exhaustion of Kupffer cells and hepatic stellate cells (HSCs) and CD4 + T cells by disrupting their MDM2p53 axis

2011;6:e23488