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[29] Patients should only access it through a registered clinical trial or under the direct supervision of an appropriately qualified healthcare professional

We posit that the surge in fetal blood IGF-1 is a compensatory response to the maternal GLP-1RA-induced fetal growth restriction

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J Clin Endocrinol Metab 2015

Increased insulin-independent glucose uptake (4) and reduced counterregulatory responses (21) may also contribute to hypoglycemia in patients with PBH

GLP-1 is also believed to participate in weight loss after bariatric surgery which brings food more rapidly into the distal small intestine
