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It increased the production of NO, which is an anti-atherogenic molecule [37, 38]
Subsets of the data based on drug and time on treatment were analyzed independently given the anomalous data with short durations of therapy

S5A), but not in Glp1r Ghsr neurons (Supplementary Fig

& Westbrook, R
3 Conventional oral hypoglycemics agents included sulfonylureas, dipeptidyl peptidase 4 inhibitors (DPP4i), sodium glucose co-transporter 2 inhibitors (SGLT2is), thiazolidinediones, or metformin
