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However, our understanding of GLP-1R expression and downstream signaling in the myocardium across species is incomplete

This suggests the possibility of another cell population that may take the lion's share of GLP1 receptor expression within the paraventricular hypothalamic nucleus or multiple other populations of neurons that have yet to be defined

doi: 10.1111/dom.12937

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In addition, NaB treatment induced GLP-1R expression in human HepG2 cells and markedly reversed the FFA-induced downregulation of GLP-1R expression but not the expression of TNF- or lipopolysaccharide in vitro (Fig

Parkinson's disease and diabetes mellitus: common mechanisms and treatment repurposing