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Specifically, after a challenge dose of alcohol, participants in the GLP-1RA group have a delayed rise in BrAC and a similar delayed rise in the subjective effects of alcohol

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GLP-1s may not cure Alzheimers as a monotherapy, but they are reshaping how we think about the interface of neurodegenerative disease etiology, metabolic health, vascular function and they likely will cement themselves as foundational tools in delaying cognitive decline across aging populations