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thus, there is a smaller window for weight loss, as body weight defence mechanisms have a greater role in lean mice, potentially confounding the absence of treatment effects

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Interestingly, GLP-1 receptors are expressed in certain areas of the central nervous system responsible for drug- and food-related rewarding effects, such as the ventral tegmental area and the nucleus accumbens [] Consistent with this, GLP-1 RAs attenuate the drug reward for substances such as nicotine in animal models [,]

Both preclinical and clinical studies suggest that GLP-1 RAs reduce systemic and central inflammation, potentially restoring immune homeostasis in brain regions critical for emotional regulation and reward processing, such as the prefrontal cortex and limbic structures [26,82,84]

Moreover, epidemiological data have established a significant association between visceral obesity and the pathogenesis of primary hepatic malignancies [15]