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Description
Strategies to correct the incretin defects in patients with type 2 diabetes include replacement of GLP-1 with a long-acting analog that resists degradation and development of drugs that inhibit the enzyme DPP-4, which breaks down GLP-1
11 A study has shown that GLP-1Rs are increased in the visceral adipose tissue of morbidly obese patients with insulin resistance
administered GLP-1 depends on vagal afferents [692,694] and thus suggests a similar mechanism for i.p
2018 doi: 10.1111/jdi.12900
Molecular Metabolism 57
No significant association of GLP1RA treatment was detected for any of those neoplasms, with low heterogeneity between studies
