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This interconnection explains why anxiety reports on GLP-1 therapy vary significantly among individuals with similar doses and treatment duration

Given that this disorder represents a model of extreme modification of glucose metabolism in response to bariatric/metabolic surgery, understanding the metabolic pathways perturbed in PBH may inform our understanding of intestinal regulation of glucose homeostasis and uncover new therapeutic targets for PBH and, more broadly, for nonsurgical approaches to improve systemic glucose metabolism

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