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After careful analysis and comparison between the four treatment groups, we found that normal CSMCs are capable of producing several cytokines and chemokines at the basal level, while inducing inflammation by LPS stimulation caused a marked increase in the expression of TNF-, IL-1, M-CSF, TCA-3, and SDF-1

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Resmetirom and GLP-1 agonists for MASH: complementary rather than exclusive Resmetirom exhibits promising results as a specific treatment for MASH

The activation of GLP-1R triggers a complex intracellular signaling cascade, which at the end activates protein kinase A (PKA) pathway via production of cyclic adenosine monophosphate (cAMP) ( GLP-1 exerts potent glucose-lowering effects through multiple mechanisms including insulin release stimulation and suppression of glucagon secretion