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Jean-Pierre Raufman , working with biochemist John Eng , discovered that the Gila monster (a desert reptile) produces in its saliva a peptide that mimics GLP-1s action but is not rapidly destroyed by DPP-4 ( [45] ) ( [46] )

GIP Receptor Agonism GIP is secreted by intestinal K cells in the proximal duodenum and, similarly to GLP-1, plays a key role in regulating post-prandial blood glucose homeostasis and lipid metabolism [23, 54]

[DOI] [PubMed] [Google Scholar] 47.Jastreboff A.M., Aronne L.J., Ahmad N.N., et al

1.2 Etiology and mechanisms Postprandial Hypoglycaemia (PPHG), also known as post-bariatric hypoglycaemia (PBH) when it occurs after bariatric surgery, is a condition where blood glucose levels drop excessively after eating [22, 23].This complex condition arises from a combination of factors, rather than a single cause, and involves a variety of interacting mechanisms that are still being investigated [23, 24]

Comparison of the Effects of Liraglutide, Tirzepatide, and Retatrutide on Diabetic Kidney Disease in Db/Db Mice