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Researchers tracked the participants' breath alcohol levels, cravings, appetite, nausea and blood sugar, and also asked, "How drunk do you feel?" on a scale of 010 over the course of four hours

This is remarkable because it means GLP-1 agonists like tirzepatide may work through a mechanism that parallels the most successful class of migraine-specific drugs ever developed

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My question: given my MS stability but significant metabolic risk factors (obesity, pre-diabetes, cholesterol, thyroid), do you think a GLP-1 agonist (such as tirzepatide or semaglutide) would be a reasonable option for me

Teede et al