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Based on these findings, this review summarizes the pathophysiological mechanisms underlying EDC, with a particular focus on how chronic inflammation contributes to its development and progression through pathways involving neurotransmitter imbalance, HPA axis dysregulation, and impaired synaptic plasticity

Mohd 1 , M

doi: 10.1016/j.urology.2003.11.009

Another complication could be that altering the capacity of a single antioxidative enzyme may produce indirect effects on ROS availability that then alter the flux through other pathways that oxidize GSH, thus masking more direct effects

doi:10.1007/s00403-017-1794-4 14